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• August 11, 2021 at 5:53 pm #30980
  Robin Rose

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  SSRP Certified

  Aloha

  I am looking at fibrosis from the perspective of chronic kidney disease – the focus being early intervention in stage2-3, peptides and bioregulators having a potent place.

  As I study this, I am repeatedly finding the whole TB4 has a resultant fibrosis, where the FRAGMENT does not.

  IMO this matters, see the studies I share below, due diligence in a prolonged chronic illness requires this awareness.  Pondering that cartoon from Tremblay about how frag 1-4 has a very different persona than 1-15, and the 17-23 frag is the mast cell effecting fragment, hair and wounds also, the 40-43 cardio focused… and then this Ac-SDKP::: does anyone USE this in their practice? I am interested in this.

  And would LOVE some intelligent discussion about this. THANKS!

  ~~~For example:: this study shows the 1-2 FRAG as protective but the 43 aa peptide to result in fibrosis:
  https://www.tandfonline.com/doi/full/10.1517/14712598.2015.1009891

  ~~~AND: this one says that TB4 itself caused# fibrosis but the tetrapeptide DEGRADATION PRODUCT Ac-SDKP Reversed IT!
  https://docs.google.com/document/d/1GKsfW1E0mS1cWUt6Axe3vsZ1KInpJ4V7jgUWXgFp2AA/edit

  August 12, 2021 at 12:50 am #30983
  Kristelle Reyes

  Member

  SSRP Staff

  Hello @byurthgmail-com, @drhusaininterlinkedmd-com and @cpaigepaigemd-com, would love to have your insights here.

  Thank you! 🙂

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