What a great topic – this is a tough mechanism – it’s from the neuroleptic induced dopamine and the nyostrragla pathway the D2 dopamine receptors, there is a inhibition of these receptors and there is an excess of dopamine that causes an excess of these abnormal movements of anti-psychotics
Traditional approaches of clonazepam and other monoamine  – in my practice, I work on DHHB it’s a Gaba-A Agonist – no tolerance, no dependancies, significant effect in neuronal health and cellular health in its pathways. Take a significant look at using this – 30mg capsules –  twice a day – you can do 3x a day in intervals.