From the video: Dr. Seeds (transcription edited for readability)
Yeah, tinnitus is a multi … definitely another autoimmune, it is an inflammatory process that does happen, it’s a different type of stress that activates the inflammasome that activates the pro inflammatory pathway, it’s like a DAMP, a damage associated molecular pattern that affects that receptor, the pattern recognition receptors, that activate inflammasomes. So, we know it has that kind of , like , an autoimmune process , but it’s an inflammasome activator , so this is where we do like working with Thymosin beta 4 and thymosin alpha 1, to try to nullify the inflammasome.  I know it’s harder to get right now, cerebrolysin is something that I add into this after I treat the immune part of this. You could look at the potentials of butyrate or ketone esters for the histone deacetylase activity, that could be helpful, again, with turning off the inflammasome. There can be many different roads to this activation. So, you might have to try a couple of different pathways, but I think that’s something that you can get to , and I always tell people to look for Dysbiosis with this too. That’s why I like working with butyrate and getting that into this process.
You’re going to go deeper and you’re going to look at cell membrane, you’re going to look at phospholipids, so it’s going to get a little complicated. You’re going to find out more and more that there’s a lot more going on with this. It’s a process where this damage associated molecular pattern, the DAMP is the main activator of the inflammasome that causes this. This is what we think at this point in time. There could be other aspects of how this is activated but that would be still from the autoimmune part. I just gave you the inflammasome aspect of this.
Great question that comes up time and time again and we’ve had success in treating this in many different ways, with TA1 by itself, with TB4 by itself, cerebrolysin by itself, with ketone esters, Ketone esters, by themselves.
I gave you kind of a way to look at going after the problem. Think about going after the inflammasome. I think that will give you more success in picking different ways of combining all those things together, which I think is probably the right way to go. And it’s not going to happen overnight either. Be prepared for up to about three months before you say, okay, what are some other options ?
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