This course teaches a cellular medicine framework for understanding post-viral fatigue through innate immune activation, energy regulation, and the biology of inflammation resolution. You’ll examine how the cGAS–STING pathway can amplify interferon and inflammatory signaling, and how redox balance and mitochondrial energetics shape symptom patterns and recovery capacity. The course also introduces specialized pro-resolving mediators (SPMs)—lipid signals derived from omega-3 and omega-6 fats—and uses them to explain how the body shifts from persistent inflammation toward resolution processes, alongside practical cautions about supplement quality and combining multiple agents. Throughout, the focus is on building clinical reasoning: interpreting mechanisms, recognizing where evidence is uncertain, and using measurement concepts (such as the omega-3 index) to support follow-up over time. This approach is designed to help clinicians plan and reassess care more systematically in heterogeneous presentations where single lab markers and one-size-fits-all strategies often fall short. It is well-suited for interdisciplinary clinicians and trainees who see complex chronic or post-viral cases and want a clearer mechanistic map for discussion, monitoring, and clinical decision-making.

The course is organized as mechanism-first teaching across two days, with recurring frameworks that connect innate immune signaling, immunometabolism, and inflammation resolution. It integrates lecture-style instruction with case-based and Q&A moments to support clinical interpretation without relying on fixed protocols.

• Over 10 hours of video content
• 2 modules
• 7 video lessons
• Downloadable learning guides